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Vasodilation is achieved by activating endothelial nitric oxide synthase (eNOS) and increasing nitric oxide (NO) production via the cAMP/PKA, AMPK, and PI 3 K/Akt signaling pathways [17]

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The pathogenesis originates from an imbalance in hepatic lipid homeostasis, where excessive triglyceride accumulation results from increased lipid influx, enhanced de novo lipogenesis, and impaired lipid export or oxidation

Diabetes 61:12501262 Nachnani JS, Bulchandani DG, Nookala A et al (2010) Biochemical and histological effects of exendin-4 (exenatide) on the rat pancreas