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Summary Clinical Presentation More common in people with darker skin Correlates with the configuration and distribution of the causative inflammatory dermatosis Causes include inflammatory conditions, cutaneous injuries, irritants, or burns Histologic Features Decrease melanin of the basal keratinocytes Upper dermal/submucosal melanophages Sparse superficial lymphohistiocytic inflammation Inflammatory cause may be coexisting Differential Diagnosis Sarcoidosis Lichen sclerosus Hypopigmented mycosis fungoides Hypopigmented extramammary Pagets disease Hypopigmentation after medication Vitiligo Chemical leukoderma Takeaway Essentials Clinical Relevant Pearls Correlates with distribution of the causative inflammatory dermatosis Pathology Interpretation Pearls Decreased melanin of the basal keratinocytes Upper dermal/submucosal melanophages Immunohistochemical/Molecular Findings Fontana-Masson special stain may show decreased melanization of basal keratinocytes Immunohistochemical melanocytic markers (MITF, HMB45, etc.) may show a decreased number of melanocytes Vitiligo Clinical Features Vitiligo, an acquired leukoderma, is one of the most frequent disorders of pigmentation of the skin and is characterized by distinct areas of skin and mucous membranes depigmentation

Most TCM studies have focused on downstream effects (e.g., upregulating Nrf2/GPX4) but fail to identify active ingredients or their direct binding partners, hindering standardized development and clinical approval

Wait at least thirty minutes before brushing to give your enamel time to reharden

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